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Hokkaido University Collection of Scholarly and Academic Papers >
Showing results 1 to 6 of 6
| Type | Author(s) | Title | Other Titles | Citation | Citation(alt) | Issue Date | | article (author version) | Tanaka, Motoki; Kawahara, Koichi; Kosugi, Tatsuro; Yamada, Takeshi; Mioka, Tetsuo | Changes in the spontaneous calcium oscillations for the development of the preconditioning-induced ischemic tolerance in neuron/astrocyte co-culture. | - | Neurochemical Research | - | Jun-2007 |
| article (author version) | Yamada, Takeshi; Kawahara, Koichi; Kosugi, Tatsuro; Tanaka, Motoki | Nitric Oxide Produced During Sublethal Ischemia Is Crucial for the Preconditioning-Induced Down-Regulation of Glutamate Transporter GLT-1 in Neuron/Astrocyte Co-Cultures | - | Neurochemical Research | - | Jan-2006 |
| article (author version) | Kosugi, Tatsuro; Kawahara, Koichi; Yamada, Takeshi; Nakajima, Takayuki; Tanaka, Motoki | Functional Significance of the Preconditioning-induced Down-regulation of Glutamate Transporter GLT-1 in Neuron/astrocyte Co-cultures | - | Neurochemical Research | - | Sep-2005 |
| article (author version) | Kawahara, Koichi; Kosugi, Tatsuro; Tanaka, Motoki; Nakajima, Takayuki; Yamada, Takeshi | Reversed operation of glutamate transporter GLT-1 is crucial to the development of preconditioning-induced ischemic tolerance of neurons in neuron/astrocyte co-cultures | - | Glia | - | Feb-2005 |
| article (author version) | Kawahara, Koichi; Yanoma, Junji; Tanaka, Motoki; Nakajima, Takayuki; Kosugi, Tatsuro | Nitric Oxide Produced During Ischemia Is Toxic but Crucial to Preconditioning-Induced Ischemic Tolerance of Neurons in Culture | - | Neurochemical Research | - | Apr-2004 |
| article (author version) | Kawahara, Koichi; Hosoya, Rui; Sato, Hideomi; Tanaka, Motoki; Nakajima, Takayuki; Iwabuchi, Sadahiro | Selective blockade of astrocytic glutamate transporter GLT-1 with dihydrokainate prevents neuronal death during ouabain treatment of astrocyte/neuron cocultures | - | Glia | - | Dec-2002 |
Showing results 1 to 6 of 6
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