The data of change in macrophage gene expression which induced by perilipin 1 overexpression

Yamamoto, Kohei; Miyoshi, Hideaki; Cho, Kyu Yong; Nakamura, Akinobu; Greenberg, Andrew S.; Atsumi, Tatsuya

The data of change in macrophage gene expression which induced by perilipin 1 overexpression

Yamamoto, Kohei; Miyoshi, Hideaki; Cho, Kyu Yong et al.

Data in Brief, 2018, 19, 179-182

Permalink : https://hdl.handle.net/2115/72098

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論文情報

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アクセス権	open access
URI	https://hdl.handle.net/2115/72098
タイトル	The data of change in macrophage gene expression which induced by perilipin 1 overexpression
著者	Yamamoto, Kohei ; Miyoshi, Hideaki ; Cho, Kyu Yong ; Nakamura, Akinobu ; Greenberg, Andrew S. ; Atsumi, Tatsuya
著者名 Yamamoto, Kohei
e-Rad_Researcher 30360902 著者名 Miyoshi, Hideaki
著者名 Cho, Kyu Yong
e-Rad_Researcher 70552420 著者名 Nakamura, Akinobu
著者名 Greenberg, Andrew S.
e-Rad_Researcher 20301905 著者名 Atsumi, Tatsuya
言語	英語
キーワード	perilipin1
	PLIN1
	Macrophage
	Atherosclerosis
発行日	2018-08
出版者	Elsevier
収録物名	Data in Brief
巻(号)	19
ページ	179-182
抄録	The data presented here are related to the research article entitled "Overexpression of Perilipin1 protects against atheroma progression in apolipoprotein E knockout mice". This paper describes data that were obtained from perilipin 1 (PLIN1) transgenic mice (Plin1Tg) regarding atherosclerosis. The main aim of collecting the data was to clarify the role of PLIN1 in the pathophysiology of atherosclerosis. The data were collected from C57BL/6J mice, apolipoprotein E knockout mice (ApoeKO) and Plin1Tg/ApoeKO. The atherosclerotic lesion areas of aorta were 3.3 ± 1.2% in C57BL/6J mice, 14.2 ± 3.2% in ApoeKO, and 5.6 ± 1.9% in Plin1Tg/ApoeKO. Body weight, gonadal adipose mass and plasma triglyceride concentrations were comparable among the three groups. Furthermore, PLIN1 overexpression did not affect the gene expressions related to cholesterol influx and efflux in macrophage.
資源タイプ	学術雑誌論文
出版タイプ	Version of Record
権利情報	Creative Commons Attribution 4.0 International
PISSN	2352-3409
関連情報 (references)	HDL https://hdl.handle.net/2115/68843
関連情報 (isIdenticalTo)	出版社版DOI https://doi.org/10.1016/j.dib.2018.05.027