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Showing results 1 to 9 of 9
TypeAuthor(s)TitleOther TitlesCitationCitation(alt)Issue Date
articleHashimoto, Shigeru; Furukawa, Shotaro; Hashimoto, Ari; Tsutaho, Akio; Fukao, Akira; Sakamura, Yurika; Parajuli, Gyanu; Onodera, Yasuhito; Otsuka, Yutaro; Handa, Haruka; Oikawa, Tsukasa; Hata, Soichiro; Nishikawa, Yoshihiro; Mizukami, Yusuke; Kodama, Yuzo; Murakami, Masaaki; Fujiwara, Toshinobu; Hirano, Satoshi; Sabe, HisatakaARF6 and AMAP1 are major targets of KRAS and TP53 mutations to promote invasion, PD-L1 dynamics, and immune evasion of pancreatic cancer-Proceedings of the National Academy of Sciences of the United States of America (PNAS)-27-Aug-2019
articleKinoshita, Rumiko; Nam, Jin-Min; Ito, Yoichi M.; Hatanaka, Kanako C.; Hashimoto, Ari; Handa, Haruka; Otsuka, Yutaro; Hashimoto, Shigeru; Onodera, Yasuhito; Hosoda, Mitsuchika; Onodera, Shunsuke; Shimizu, Shinichi; Tanaka, Shinya; Shirato, Hiroki; Tanino, Mishie; Sabe, HisatakaCo-Overexpression of GEP100 and AMAP1 Proteins Correlates with Rapid Local Recurrence after Breast Conservative Therapy-Plos one-7-Oct-2013
articleMenju, Toshi; Hashimoto, Shigeru; Hashimoto, Ari; Otsuka, Yutaro; Handa, Haruka; Ogawa, Eiji; Toda, Yoshinobu; Wada, Hiromi; Date, Hiroshi; Sabe, HisatakaEngagement of Overexpressed Her2 with GEP100 Induces Autonomous Invasive Activities and Provides a Biomarker for Metastases of Lung Adenocarcinoma-PLoS One-22-Sep-2011
articleOtsuka, Yutaro; Oikawa, Tsukasa; Yoshino, Hinako; Hashimoto, Shigeru; Handa, Haruka; Yamamoto, Hiroki; Hashimoto, Ari; Sabe, HisatakaFrequent overexpression of AMAP1, an Arf6 effector in cell invasion, is characteristic of the MMTV-PyMT rather than the MMTV-Neu human breast cancer model-Cell communication and signaling-5-Jan-2018
articleOikawa, Tsukasa; Otsuka, Yutaro; Onodera, Yasuhito; Horikawa, Mei; Handa, Haruka; Hashimoto, Shigeru; Suzuki, Yutaka; Sabe, HisatakaNecessity of p53-binding to the CDH1 locus for its expression defines two epithelial cell types differing in their integrity-Scientific reports-25-Jan-2018
articleHashimoto, Ari; Oikawa, Tsukasa; Hashimoto, Shigeru; Sugino, Hirokazu; Yoshikawa, Ayumu; Otsuka, Yutaro; Handa, Haruka; Onodera, Yasuhito; Nam, Jin-Min; Oneyama, Chitose; Okada, Masato; Fukuda, Mitsunori; Sabe, HisatakaP53-and mevalonate pathway-driven malignancies require Arf6 for metastasis and drug resistance-Journal of cell biology-11-Apr-2016
article (author version)Tenma, Taro; Mitsuyama, Hirofumi; Watanabe, Masaya; Kakutani, Naoya; Otsuka, Yutaro; Mizukami, Kazuya; Kamada, Rui; Takahashi, Masayuki; Takada, Shingo; Sabe, Hisataka; Tsutsui, Hiroyuki; Yokoshiki, HisashiSmall-conductance Ca2+-activated K+ channel activation deteriorates hypoxic ventricular arrhythmias via CaMKII in cardiac hypertrophySK channels deteriorate hypoxic ventricular arrhythmiaAmerican journal of physiology. Heart and circulatory physiology-Aug-2018
theses (doctoral)大塚, 勇太郎癌悪性度進展の分子基盤の解明Molecular Bases of The Cancer Malignancy Development--22-Mar-2018
theses (doctoral - abstract of entire text)大塚, 勇太郎癌悪性度進展の分子基盤の解明 [全文の要約]Molecular Bases of The Cancer Malignancy Development [an abstract of entire text]--22-Mar-2018
Showing results 1 to 9 of 9

 

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