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通常型膵癌の浸潤、転移メカニズムに関する分子生物学的研究

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Please use this identifier to cite or link to this item:http://doi.org/10.14943/doctoral.k14320
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Title: 通常型膵癌の浸潤、転移メカニズムに関する分子生物学的研究
Other Titles: The molecular biological studies on the mechanisms of invasion and metastasis of pancreatic ductal adenocarcinoma
Authors: 古川, 聖太郎 Browse this author
Issue Date: 25-Dec-2020
Conffering University: 北海道大学
Degree Report Number: 甲第14320号
Degree Level: 博士
Degree Discipline: 医学
Examination Committee Members: (主査) 教授 武冨 紹信, 教授 園下 将大, 教授 近藤 亨, 教授 田中 伸哉
Degree Affiliation: 医学研究科(医学専攻)
(Relation)haspart: Shigeru Hashimoto1, Shotaro Furukawa1, Ari Hashimoto1, Akio Tsutaho, Akira Fukao, Yurika Sakamura, Gyanu Parajuli, Yasuhito Onodera, Yutaro Otsuka, Haruka Handa, Tsukasa Oikawa, Soichiro Hata, Yoshihiro Nishikawa, Yusuke Mizukami, Yuzo Kodama, Masaaki Murakami, Toshinobu Fujiwara, Satoshi Hirano and Hisataka Sabe. 1Shigeru Hashimoto, Shotaro Furukawa and Ari Hashimoto contributed equally to this work. ARF6 and AMAP1 are major targets of KRAS and TP53 mutations to promote invasion, PD-L1 dynamics, and immune evasion of pancreatic cancer. Proceedings of the National Academy of Sciences of the United States of America, 2019 Aug 27;116(35):17450-17459
Type: theses (doctoral)
URI: http://hdl.handle.net/2115/80220
Appears in Collections:課程博士 (Doctorate by way of Advanced Course) > 医学院(Graduate School of Medicine)
学位論文 (Theses) > 博士 (医学)

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